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Результаты поиска “Arthritis pathophysiology concept mapping”
Rheumatoid Arthritis Pathophysiology
 
15:34
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Просмотров: 285116 Armando Hasudungan
RA Concept Maps Mind Express
 
01:37
Просмотров: 32 Phyl Macomber
Asthma Pathophysiology
 
10:30
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Просмотров: 350728 Armando Hasudungan
Pathophysiology of Gastroesophageal Reflux Disease (GERD)
 
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Physiology lecture on gastroesophageal reflux disease including causes based on the anatomy of the gastroesophageal junction.
Просмотров: 42592 Andrew Wolf
Omega-3 fatty acids and inflammatory processes by Philip Calder
 
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Webcast of the presentation entitled 'Omega-3 fatty acids and inflammatory processes: From molecules to man' given by Philip Calder (University of Southampton, United Kingdom) at the joint Biochemical Society/British Society for Immunology Hot Topic Event 'Nutrient mechanisms in inflammation, infection and immunity'.
Просмотров: 11827 Biochemical Society
Carbohydrates & sugars - biochemistry
 
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What are carbohydrates & sugars? Carbohydrates simple sugars as well as complex carbohydrates and provide us with calories, or energy. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Просмотров: 55417 Osmosis
Pathophysiology of Pyleonephritis
 
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Просмотров: 322 Derek Devino
Glomerular diseases
 
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This is a summary of diseases that affect the glomerulus of the kidney, including those that case nephrotic and nephritic syndromes. I created this presentation with Google Slides. Image were created or taken from Wikimedia Commons I created this video with the YouTube Video Editor. ADDITIONAL TAGS: Glomerular diseases Minimal change disease Focal segmental glomerulosclerosis Membranous glomerulonephritis Diabetic nephropathy Amyloid nephropathy Lupus nephritis Membranoproliferative glomerulonephritis IgA nephropathy Acute postinfectious glomerulonephritis Anti-GBM disease ANCA glomerulonephritis Nephrotic Nephritic Systemic Primary glomerular disease with nephrotic syndrome Most common in children, especially young children LM: “minimal change” -- looks normal IF: negative (no immune complex deposition) EM: foot process effacement, see figure secondary causes: cancer, infection, drugs, atopy (hyperallergic) Associated with lymphoma and use of NSAIDs Primary glomerular disease with nephrotic syndrome Focal means some glomeruli, segmental means not all of glomerulus LM: segmental glomerular scarring, see hyaline material (deposits from plasma) IF: positive for Ig and complement, granular appearance EM: segmental effacement Primary FSGS is idiopathic Secondary causes: genetic, infection (HIV, parvovirus), drugs (heroin), sickle cell, obesity Most common in blacks Primary glomerular disease with nephrotic syndrome LM: capillary wall thickening with IgG and C3 immune complexes IF: positive for IgG and C3 in the capillary, granular EM: immune complexes in subEPIthelial space Primary cause: Ab against anti-phospholipase A2 receptor Secondary causes: cancer, lupus, NSAIDs, HBV, Hep B, syphilis… Ag can be Hep B or cancer Secondary nephropathy with nephrotic syndrome; DM is primarily systemic disease Caused by DM types I and II → accumulation of glycosylated plasma protein in GBM and mesangium LM: mesangial expansion and KW nodules IF: linear staining of IgG EM: thickened GBM Treatment is reduce blood sugar, lower blood pressure, avoid nephrotoxins Most common cause for end stage renal disease in United States Secondary nephropathy with nephrotic syndrome; amyloidosis is primarily a systemic disease Caused by accumulation of polypeptides (especially AL and AA amyloid) LM: thickening of mesangium amorphous pale pink stuff in glomerulus, confirm with Congo Red stain IF: monoclonal staining of accumulated amyloid protein EM: randomly arranged fibrils Associated with rheumatoid arthritis and multiple myeloma Ranked by class: I (best) to VI (worst) Antigen antibody complexes deposit in gloms, activate complement which leads to proliferation of mesangium and infiltration of PMNs LM: endocapillary and mesangial proliferation and sometimes crescents IF: granular pattern, everything lights up! “full house pattern” EM: deposits anywhere and everywhere Hypocomplementemia - both C3 and C4 blood levels are low Presents with nephritic and/or nephrotic syndrome; kind of in between Type I is immune complex and C3 deposits Caused by bacterial infection, hep C infection, malignancies Type II is just C3 deposits but no Ig Caused by complement dysregulation LM: capillary wall thickening with hypercellularity IF: immune complexes and/or C3 granular deposits causing hypocomplementemia EM: same deposits (subendo and BM) Deposits of IgA alone or with other Igs, in mesangium Activates complement, which causes proliferation of mesangial cells LM: mesangial hypercellularity IF: IgA positive, granular pattern in the mesangium EM: mesangial deposits Primary IgA nephropathy is idiopathic Can be part of systemic disease IgA vasculitis; related to Henoch– Schönlein purpura Occurs few weeks after infection; most often follows strep or staph LM: endocapillary and mesangial hypercellularity and PMNs IF: pos for C3 in capillary walls, granular EM: subepithelial humps Hypocomplementemia; low C3 levels Treatment is supportive Nephritic Caused by autoAb to glomerular basement membrane Abs recruit complement and lymphocytes → damages capillary → proliferation and accumulation in Bowman’s space → crescent LM: crescent formation IF: pos for IgG in linear pattern EM: normal Disease is called Goodpasture’s syndrome when presents with both kidney and lung involvement LM: crescents, necrosis IF: pauci-immune; not much lights up EM: normal Associated diseases: Granulomatosis with polyangiitis (GPA); Wegener's; PR3 Eosinophilic granulomatosis with polyangiitis (eGPA); Churg–Strauss churg Strauss Microscopic polyangiitis (MPA)
Просмотров: 19897 MedLecturesMadeEasy
Burns (DETAILED) Overview - Types, Pathophysiology, TBSA
 
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Where do I get my information from: http://armandoh.org/dig Facebook: https://www.facebook.com/ArmandoHasudungan Support me: http://www.patreon.com/armando Instagram: http://instagram.com/armandohasudungan Twitter: https://twitter.com/Armando71021105 SPECIAL THANKS: Patreon members
Просмотров: 39964 Armando Hasudungan
Lupus Careplan Presentation Unit 8
 
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Description
Просмотров: 70 Teresa Barnes
Peptic ulcers - causes, symptoms, diagnosis, treatment, pathology
 
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What are peptic ulcers? Peptic ulcers are breaks in the lining of the stomach, small intestine, or esophagus. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Просмотров: 266128 Osmosis
(Bacterial) Meningitis Pathophysiology
 
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Просмотров: 51535 Armando Hasudungan
Diane Mathis (Harvard) 1: An Introduction to T Cell Tolerance
 
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https://www.ibiology.org/immunology/t-cell-tolerance/ Part 1: Introduction to T Cell Tolerance: When immunological tolerance fails, autoimmune diseases such as diabetes result. Mathis explains how T cell tolerance develops. Part 2: Transcription Factor Aire Orchestrates T Cell Tolerance: Mutation in the transcription factor Aire results in autoimmune attacks on numerous organs. Mathis tells us how Aire determines T cell tolerance. Talk Overview: To successfully fight off microbial infections, our immune systems must recognize a broad and diverse array of peptides. Occasionally, this results in the recognition of self-peptides and the development of autoimmune disease. For example, if a T cell recognizes insulin, type-1 diabetes may result. In her first talk, Dr. Mathis explains how the body has developed multiple mechanisms of immunological tolerance to prevent self-recognition. She focuses in greater detail on two particular types of T cell tolerance: clonal deletion and suppression. Patients with Autoimmune Polyglandular Syndrome Type-1, a disease that manifests as autoimmune attacks on many organs, are known to have a mutation in the transcription factor Aire. Aire is expressed in a small subset of cells in the thymus, so how does it cause autoimmune disease in so many tissues? In Part 2, Dr. Mathis describes experiments from her lab showing that Aire regulates the transcription of many self-antigens in the thymus. Expression of these self-antigens is required for the development of T cell tolerance; if Aire is mutated, autoimmunity results. Speaker Biography: Diane Mathis is a Professor in the Division of Immunology and the Department of Microbiology and Immunobiology at Harvard Medical School. She is also a principal member of the Harvard Stem Cell Institute and an associate member of the Broad Institute. Mathis’ lab studies the genetic, cellular and molecular mechanisms that determine immunological tolerance, and autoimmune diseases such as type-1 diabetes and autoimmune polyglandular syndrome that result when immunological self-tolerance fails. The lab focuses particularly on T cell tolerance. Learn more about Mathis’ research here: http://cbdm.hms.harvard.edu/research.htm Mathis’ scientific excellence has been recognized by election to the US National Academy of Sciences, the German National Academy of Sciences Leopoldina, and the American Academy of Arts and Sciences. Mathis is also an active member of the scientific community reviewing for numerous journals, organizing meetings, and teaching. She was chosen as the 2017 recipient of the FASEB Excellence in Science Award. Diane Mathis received her BSc from Wake Forest University and her PhD from the University of Rochester. She was a post-doctoral fellow at the Laboratoire de Génétique Moléculaire des Eucaryotes in Strasbourg, France and at Stanford University.
Просмотров: 6210 iBiology
Electrolyte Imbalance Signs & Symptoms: Sweet and Simple
 
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www.tootRN.com Instagram: tootRN Popular players in nursing lessons— Sodium, potassium, calcium and magnesium. Learn the major s/s of hypo- and hyper- states.
Просмотров: 132547 tootRN, LLC.
Crohn's Disease Nursing | Crohns Symptoms, Pathophysiology, Treatment, Diet NCLEX
 
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Crohn's disease nursing review regarding diet, symptoms, pathophysiology, nursing interventions and treatment for NCLEX exam. Crohn's disease is a type of inflammatory bowel disease that causes inflammation and ulcers formation in the GI tract. Crohn's can be found in both the small and large intestine; however, it seems to be most common in the terminal of the ileum and beginning of the colon. Crohn's disease affects the whole bowel lining which is why its complications are unique when comparing crohn's disease vs ulcerative colitis. Crohn's disease complications include fistulas, fissures, abscesses, malnourishment, strictures which can lead to bowel obstructions, and other changes such as mouth ulcers, skin changes, gall and liver problems etc. Crohn's disease symptoms include abdominal pain (which tends to be located in the right lower abdomen), ulcers in the mouth and GI lining, anal fissures, diarrhea which can contain pus, mucous, and blood, and undernourishment. Crohn's disease tends to present in scattered patches throughout the GI tract where healthy areas will be noted next to diseased areas. This will give a cobble-stone appearance during a scope of the GI lining. This is different than ulcerative colitis which tends to start in the rectum and migrate in a continuous fashion through the large intestine without any skipped areas. Nursing interventions for Crohn's Disease include: educating patient about this condition along with medication and diet regime and smoking cessation, monitoring nutrition status, assessing GI system by monitoring bowel sounds, bowel movements, monitoring administration of TPN (total parenteral nutrition) if ordered, providing ostomy education (if the patient has one). Medications used to treat Crohn's includes: 5-Aminosalicylates, corticosteroids, Immunosuppression drugs, etc. Quiz on Crohn's Disease: http://www.registerednursern.com/crohns-disease-nclex-questions/ Lecture Notes: http://www.registerednursern.com/crohns-disease-nclex-review/ Ulcerative Colitis NCLEX Review: https://www.youtube.com/watch?v=ICkNjkg6BHs GI NCLEX Lectures: https://www.youtube.com/playlist?list=PLQrdx7rRsKfVVQ5TeRY7Vy1Tq3pcGTDFO Subscribe: http://www.youtube.com/subscription_center?add_user=registerednursern Nursing School Supplies: http://www.registerednursern.com/the-ultimate-list-of-nursing-medical-supplies-and-items-a-new-nurse-student-nurse-needs-to-buy/ Nursing Job Search: http://www.registerednursern.com/nursing-career-help/ Visit our website RegisteredNurseRN.com for free quizzes, nursing care plans, salary information, job search, and much more: http://www.registerednursern.com Check out other Videos: https://www.youtube.com/user/RegisteredNurseRN/videos All of our videos in a playlist: https://www.youtube.com/watch?v=pAhHxt663pU&list=PLQrdx7rRsKfXMveRcN4df0bad3ugEaQnk Popular Playlists: NCLEX Reviews: https://www.youtube.com/playlist?list=PLQrdx7rRsKfWtwCDmLHyX2UeHofCIcgo0 Fluid & Electrolytes: https://www.youtube.com/playlist?list=PLQrdx7rRsKfWJSZ9pL8L3Q1dzdlxUzeKv Nursing Skills: https://www.youtube.com/playlist?list=PLQrdx7rRsKfUhd_qQYEbp0Eab3uUKhgKb Nursing School Study Tips: https://www.youtube.com/playlist?list=PLQrdx7rRsKfWBO40qeDmmaMwMHJEWc9Ms Nursing School Tips & Questions" https://www.youtube.com/playlist?list=PLQrdx7rRsKfVQok-t1X5ZMGgQr3IMBY9M Teaching Tutorials: https://www.youtube.com/playlist?list=PLQrdx7rRsKfUkW_DpJekN_Y0lFkVNFyVF Types of Nursing Specialties: https://www.youtube.com/playlist?list=PLQrdx7rRsKfW8dRD72gUFa5W7XdfoxArp Healthcare Salary Information: https://www.youtube.com/playlist?list=PLQrdx7rRsKfVN0vmEP59Tx2bIaB_3Qhdh New Nurse Tips: https://www.youtube.com/playlist?list=PLQrdx7rRsKfVTqH6LIoAD2zROuzX9GXZy Nursing Career Help: https://www.youtube.com/playlist?list=PLQrdx7rRsKfVXjptWyvj2sx1k1587B_pj EKG Teaching Tutorials: https://www.youtube.com/playlist?list=PLQrdx7rRsKfU-A9UTclI0tOYrNJ1N5SNt Personality Types: https://www.youtube.com/playlist?list=PLQrdx7rRsKfU0qHnOjj2jf4Hw8aJaxbtm Dosage & Calculations for Nurses: https://www.youtube.com/playlist?list=PLQrdx7rRsKfUYdl0TZQ0Tc2-hLlXlHNXq Diabetes Health Managment: https://www.youtube.com/playlist?list=PLQrdx7rRsKfXtEx17D7zC1efmWIX-iIs9
Просмотров: 119934 RegisteredNurseRN
l2 glomerulonephritis
 
01:02:17
Просмотров: 136 تعلم الطب
Rheumatic Fever & Heart Disease - Part 1/7
 
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Watch 800+ Medical Lectures at https://www.DrNajeebLectures.com ─────────────── DR. NAJEEB LECTURES ─────────────── Dr. Najeeb Lectures are the World's Most Popular Medical Lectures. Over 1 Million+ students from 190 countries trust Dr. Najeeb Lectures to Master Medical Sciences. Sign up for a membership plan on our website and access 800+ videos on Basic Medical Sciences & Clinical Medicine. ───────────────── OUR YOUTUBE CHANNEL ───────────────── Here on YouTube, we only upload free sample videos. Most of them are teaser videos (not complete lectures). If you like these videos you can check out our entire video library on our website at https://www.DrNajeebLectures.com. ────────────────────── WHY SIGN UP FOR MEMBERSHIP? ────────────────────── ► 800+ Medical Lectures. ► Basic Medical Sciences. ► Clinical Medicine. ► New videos every week in HD. ► Download videos for offline access. ► Fast video playback (0.5x - 2x) ► Watch videos on any device. ► Fanatic customer support. ► Trusted by 1 Million+ students. Learn more at https://www.DrNajeebLectures.com
Просмотров: 225902 Dr. Najeeb Lectures
Osteoporosis Screening Guidelines *USMLE STEPs 1, 2 3*
 
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Super high-yield Osteoporosis Screening Guidelines for the USMLE STEP exams, as well as for your clinical practice! Know more, do better! Good luck to you all! We are here for you! About the USMLE CHANNEL - We are Niya and Raj, and we are here to share with you our passion for Medicine! We have successfully passed all USMLE STEPs and we know what YOU NEED to know, in order to SCORE phenomenally HIGH and enjoy RESIDENCY in the UNITED STATES. We are creating medical content videos for all STEPs - yes ALL of them-1, 2 and 3! Our videos are finely tuned, to provide you with the complete understanding of the most updated material, that will help you pick the right answers on the tests and in your future clinical practice. Because what you are studying here, trust us, you will be needing during your residency training! In our videos, we will be using our very own PERSONALLY CRAFTED MIND MAPS, that will make all that large content seem small and make concepts stick. We'll also be integrating quite a lot, which in our years of teaching experience has proven to give the best results. We have walked your journey, and if when starting it, we knew what we know now, life would have been so much easier. And that’s the idea behind the birth of this channel, to be here for you and give you the knowledge that will help you succeed. Enjoy our videos! Stay updated! Love what you are learning! Because in the end - IT IS ALL WORTH IT! DISCLAIMER: The Video Content has been made available for informational and educational purposes only. The Video Content is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Always seek the advice of qualified health professionals and educators for clarification or continued education on any materials expressed herein prior to patient treatment and skills implementation. Attributions: Music -https://www.bensound.com Photo by Aaditya Arora from Pexels
Просмотров: 540 THE USMLE CHANNEL.
Immunology: RA
 
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Immunology: RA
Просмотров: 564 Rebecca Smith
Foods to Cure Pyelonephritis | Including Liquids, Antioxidants & Omega 3 Rich Foods
 
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Don't forget to check out our brand new website - http://bit.ly/hmvnutr Be it any condition, a healthy diet is a road to fast recovery. Watch this video to learn more about the food groups to consume to help you get rid of Pyelonephritis! ----------------------------- Pyelonephritis Pyelonephritis is essentially a complication of a typical bladder infection. It’s a bacterial infection of the urinary tract that doesn't limit itself to the bladder & the urethra, but also extends up to involve the kidney & ureters. This infection may also spread to the blood. ----------------------------- Symptoms: • Fever • Nausea • Back pain • Abdominal pain • Frequent urge to urinate • Burning sensation while urination • Visibility of pus or blood in the urine ----------------------------- Food groups to consume in pyelonephritis: 1. Liquids: Flush out the bacteria from the kidneys Foods to consume: Water, green vegetable or fruit juices, herbal teas, soups, and buttermilk 2. Antioxidants: Defend the body against free radicals & help fight infection Foods to consume: Papaya, oranges, pomegranate, plums, peaches, mangoes, carrots, beetroot, spinach, broccoli, blueberries, raspberries & strawberries 3. Omega 3: They have anti-inflammatory properties Foods to consume: Mackerel, herring, trout, salmon, tuna, olive, and other vegetable oils and nuts ----------------------------- Tips: • Avoid consuming alcohol, coffee, carbonated drinks, fried foods, processed foods, pickles, sauces and any excessively salty foods ----------------------------- SUBSCRIBE TO HOMEVEDA NUTRITION: http://www.youtube.com/user/homevedanutrition?sub_confirmation=1 JOIN US ON FACEBOOK: http://www.facebook.com/homeveda FOLLOW US ON TWITTER: http://twitter.com/homeveda ----------------------------- The above recipes have been developed in consultation with a nutritionist. However, these are not intended as a substitute for the medical advice of a trained health professional. All matters regarding your health require medical supervision. Consult your physician before adopting the suggestions in this video, as well as about any condition that may require diagnosis or medical attention. Refer to the terms of use on our website http://www.homeveda.com.
Просмотров: 1050 HomeVeda Nutrition
Postinfectious (Post-streptococcal) Glomerulonephritis Illness Script - USMLE, Medicine, Pediatrics
 
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Overview of classic presentation and illness script for postinfectious glomerulonephritis (also known poststreptococcal glomerulonephritis). Please like and subscribe if you like this video as I plan to regularly upload more videos with ideas you leave in the comments! Follow me on Twitter: https://twitter.com/illness_scripts Use my channel to help study for USMLE step 1, USMLE Step 2, USMLE Step 3, COMPLEX, NCLEX, and other board exams including Internal Medicine, Family Medicine, and Pediatrics among others. Pencast Post infectious glomerulonephritis Poststreptococcal glomerulonephritis Granulomatosis with polyangiitis (Wegener's) Microscopic polyangiits IgA nephropathy Goodpasture's Illness Scripts Medical Education Medicine Pediatrics USMLE COMPLEX NCLEX Transcript: Welcome back to Illness Scripts Pencast. Today we will be discussing another great topic frequently encountered on board exams: postinfectious glomerulonephritis. When breaking down the illness script for postinfectious GN, you need to understand the two different levels a question could be asked. The first is to have you simply identify that a glomerulonephritis in general is the most likely cause of kidney injury in your patient. The next level is questions that make you differentiate between the types of glomerulonephritis (or GN). Predisposing factors are less specific in this disorder but more commonly seen in children than adults (but not as skewed as some other disorders so do not exclude this diagnosis if the patient is an adult). As the name suggests, this GN occurs after an infection. The most commonly associated infection is group A strep but staph and gram negative organisms can result in this GN. These patients will often report a preceding infection at least one week prior (up to 6 weeks prior in cases of strep skin infections) to current presentation. This distinction is one of the most important of the history because IgA nephropathy can present similarly but often more concomitant with the infection. The staining with also be markedly different on biopsy but we’ll get to that. The next part of the presentation will be the part common to many of the GNs: edema, and hematuria. This case is an example where an absence of a history finding is important. In two other causes of GN, pulmonary findings are often part of the most common (or rather classic) presentation: granulomatosis with polyangiitis (formerly called Wegener’s granulomatosis) and Goodpasture’s disease (type of anti-glomerular basement member or anti-GBM disease). Both of these types of GN are not likely to occur in children as well. Exam findings will show hypertension and edema. The triad of HTN, edema, and hematuria is quite classic for GN. Keep in mind you are not likely to see evidence of active infection as by the time of presentation it is at least one week since the inciting infection. Lab findings will often provide you with complement levels to help clinch the diagnosis. In postinfectious GN, C3 is low and C4 is normal. C3 is part of the classical and alternative complement pathways but C4 is only in the classical pathway which is not activated in postinfectious GN. Thus C3 is converted to the active (or cleaved) form C3a thus decreasing measured levels of C3. The classic pathway is not involved so C4 levels remain normal. In IgA nephropathy, C3 and C4 remain normal. Urine is also characteristic for GN with RBC casts and/or dysmorphic RBCs (while these are highly suggestive of GN, don’t exclude if absent). Anti-streptolysin O (ASO) antibodies are often positive if the preceding infection was strep (but don’t exclude the diagnosis if this is negative since the infection could have been non-strep). This would be a good time to briefly review the different types of GN. The major categories are immune-complex mediated, pauci-immune, and anti-GBM. These categories are named based on the staining characteristics under microscopy when stained for different immune markers. The major immune-complex mediated GNs are postinfectious, IgA, SLE, cyroglobulinemia, and membranoproliferative GN. Pauci-immune means there are few or no immune complexes and the two majors types are granulomatosis with polyangitis (GPA) and microscopic polyangiitis (MPA). As we discussed before, the major anti-GBM GN to consider is Goodpasture’s. Thus, an ideal illness script for postinfectious GN would read: a 10-year-old boy with strep throat 1 week ago presents with diffuse edema, gross hematuria found to be hypertensive, edematous with RBC casts and dysmorphic RBCs in urine sediment with serum C3 levels decreased and C4 levels normal. Renal biopsy shows subepithelial deposits on electronic microscopy which stain positive for C3 on immunofluorescence.
Просмотров: 3488 Illness Scripts
Urinary Tract Infection Nursing NCLEX | UTI Symptoms Treatment Cystitis, Pyelonephritis, Urethritis
 
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Urinary tract infection (UTI) nursing management of the treatment, signs and symptoms and causes for the following conditions: cystitis, pyelonephritis, urethritis, ureteritis. A urinary tract infection is an infection found within the urinary tract which is mainly caused by a bacteria (usually E.coli from the GI tract). It can also be caused by a virus or fungus as well. A UTI can be found anywhere through the urinary system such as:, Lower urinary system: Urethra (Urethritis) and Bladder (Cystitis) or the upper urinary system: Ureters (ureteritis) which is usually associated with bladder or kidney infection and Kidneys (pyelonephritis). There are many defense systems in the urinary tract that help prevent a urinary tract infection. However, if these defense systems are broken down a urinary tract infection can develop. These defense system include: physiology of how urine drains out of the kidneys (no back flowing), urine itself (sterile and has antiseptic properties), lining of the urinary tract (immune cells to fight infection), prostate gland (secrete antimicrobial fluid), and normal flora of the vagina (good bacteria to fight bad bacteria). What are the causes of a urinary tract infection (UTI)? The following can cause a UTI: Hormone changes: pregnancy, menopause, birth control (changes the normal flora in the vagina), Antibiotics: changes the normal flora of the body, Renal Stones: cause blockage..urine stays in the kidneys and can’t drain, Diabetes: compromised circulation, the immune system is not able to work as strong as it should to fight the infection, high glucose in the urine (breeding ground for bacteria to flourish), bladder doesn’t empty as it should (urinary retention from nerve damage), Toiletries: excessive bubble baths, powders, perfumes, especially scented tampons and sanitary napkins, Obstructive prostatic hypertrophy…seen in male with BPH: The urethra which is surrounded by the prostate gland becomes squeezed shut from the large prostate gland which causes urine to stay in the bladder longer and the prostate gland isn’t able to properly secrete fluid that has antimicrobial properties, Vesicoureteral reflux: most common in pediatric patients and it causes urine from the bladder to back flow into the kidneys (usually congenital defect) due to a ureter valve problem, Overextended bladder, Indwelling catheters or invasive procedures, Intercourse, especially with the usage of spermicides, incontinence of the bowel, and Decreased immune system. Symptoms of urinary tract infection include: Pain when voiding (burning), Persistent need to void but not a lot is voided or can’t void, Odor to urine that is dark and cloudy (can have blood), Cramping in abdomen or pain at the costovertebral angle, Spasms of the bladder or urethra, Fever, Increased WBC on U/A Nursing management of UTI includes: assessing for signs and symptoms of UTI (catch it before it becomes complicated), Maintain fluid status (intake and output), control pain, monitor for complications, administer medications per MD order, EDUCATE the patient about how to prevent and follow MD treatment plan for UTI etc. Quiz on Urinary Tract Infection: http://www.registerednursern.com/urinary-tract-infection-nclex-questions/ Lecture Notes: http://www.registerednursern.com/urinary-tract-infection-nclex-review/ More Renal Lectures: https://www.youtube.com/playlist?list=PLQrdx7rRsKfXkfh1nWrtxLcRTrohj41su Subscribe: http://www.youtube.com/subscription_center?add_user=registerednursern Nursing School Supplies: http://www.registerednursern.com/the-ultimate-list-of-nursing-medical-supplies-and-items-a-new-nurse-student-nurse-needs-to-buy/ Visit our website RegisteredNurseRN.com for free quizzes, nursing care plans, salary information, job search, and much more: http://www.registerednursern.com Check out other Videos: https://www.youtube.com/user/RegisteredNurseRN/videos All of our videos in a playlist: https://www.youtube.com/watch?v=pAhHxt663pU&list=PLQrdx7rRsKfXMveRcN4df0bad3ugEaQnk Popular Playlists: NCLEX Reviews: https://www.youtube.com/playlist?list=PLQrdx7rRsKfWtwCDmLHyX2UeHofCIcgo0 Fluid & Electrolytes: https://www.youtube.com/playlist?list=PLQrdx7rRsKfWJSZ9pL8L3Q1dzdlxUzeKv Nursing Skills: https://www.youtube.com/playlist?list=PLQrdx7rRsKfUhd_qQYEbp0Eab3uUKhgKb Nursing School Study Tips: https://www.youtube.com/playlist?list=PLQrdx7rRsKfWBO40qeDmmaMwMHJEWc9Ms Nursing School Tips & Questions" https://www.youtube.com/playlist?list=PLQrdx7rRsKfVQok-t1X5ZMGgQr3IMBY9M Teaching Tutorials: https://www.youtube.com/playlist?list=PLQrdx7rRsKfUkW_DpJekN_Y0lFkVNFyVF Types of Nursing Specialties: https://www.youtube.com/playlist?list=PLQrdx7rRsKfW8dRD72gUFa5W7XdfoxArp Healthcare Salary Information: https://www.youtube.com/playlist?list=PLQrdx7rRsKfVN0vmEP59Tx2bIaB_3Qhdh New Nurse Tips: https://www.youtube.com/playlist?list=PLQrdx7rRsKfVTqH6LIoAD2zROuzX9GXZy
Просмотров: 128805 RegisteredNurseRN
Anemia of chronic disease | Hematologic System Diseases | NCLEX-RN | Khan Academy
 
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Created by Nauroz Syed. Watch the next lesson: https://www.khanacademy.org/test-prep/nclex-rn/hematologic-system-diseases/rn-iron-deficiency-anemia-and-anemia-of-chronic-disease/v/chronic-disease-vs-iron-deficiency-anemia?utm_source=YT&utm_medium=Desc&utm_campaign=Nclex-rn Missed the previous lesson? https://www.khanacademy.org/test-prep/nclex-rn/hematologic-system-diseases/rn-iron-deficiency-anemia-and-anemia-of-chronic-disease/v/iron-deficency-anemia-diagnosis?utm_source=YT&utm_medium=Desc&utm_campaign=Nclex-rn NCLEX-RN on Khan Academy: A collection of questions from content covered on the NCLEX-RN. These questions are available under a Creative Commons Attribution-NonCommercial-ShareAlike 3.0 United States License (available at http://creativecommons.org/licenses/by-nc-sa/3.0/us/). About Khan Academy: Khan Academy offers practice exercises, instructional videos, and a personalized learning dashboard that empower learners to study at their own pace in and outside of the classroom. We tackle math, science, computer programming, history, art history, economics, and more. Our math missions guide learners from kindergarten to calculus using state-of-the-art, adaptive technology that identifies strengths and learning gaps. We've also partnered with institutions like NASA, The Museum of Modern Art, The California Academy of Sciences, and MIT to offer specialized content. For free. For everyone. Forever. #YouCanLearnAnything Subscribe to Khan Academy’s NCLEX-RN channel: https://www.youtube.com/channel/UCDx5cTeADCvKWgF9x_Qjz3g?sub_confirmation=1 Subscribe to Khan Academy: https://www.youtube.com/subscription_center?add_user=khanacademy
Просмотров: 97922 khanacademymedicine
What treatments are available for MPGN/DDD/C3G? MPGN and C3 Glomerulopathy
 
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What treatments are available for MPGN/DDD/C3G? from the MPGN and C3 Glomerulopathy conference 2013. Guest speaker Professor Tim Goodship, Professor of Renal Medicine, Newcastle University
Просмотров: 2296 MPGN and C3 Glomerulopathy
Acute Glomerulonephritis Nursing (Poststreptococcal) | Nephritic Syndrome NCLEX Review
 
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Acute glomerulonephritis nursing (poststreptococcal) NCLEX review. Poststreptococcal glomerulonephritis is a type of nephritic syndrome which means there is the leakage of both red blood cells and proteins into the urine due to an inflamed glomerulus. This type of acute glomerulonephritis tends to present 14 days after a strep infection of the throat or skin and mainly affects the pediatric population from ages 2-10. Signs and symptoms of glomerulonephritis includes hematuria (tea or cola-colored urine), mild proteinuria, mild edema (specifically in the face and around the eyes), decreased glomerular filtration rate, increased BUN and creatinine, oliguria (low urine output), hypertension. Nursing interventions for acute glomerulonephritis includes daily weights (this is the earliest signs of fluid retention), monitoring vital signs (especially the blood pressure due to hypertension), implementing a sodium and fluid restriction (potassium restriction may be ordered if urinary output is low), administering diuretics and antihypertensives per MD order etc. Quiz on Glomerulonephritis: http://www.registerednursern.com/glomerulonephritis-nclex-questions/ Notes: http://www.registerednursern.com/acute-glomerulonephritis-poststreptococcal-nclex-review/ More Renal Lectures: https://www.youtube.com/playlist?list=PLQrdx7rRsKfXkfh1nWrtxLcRTrohj41su Subscribe: http://www.youtube.com/subscription_center?add_user=registerednursern Nursing School Supplies: http://www.registerednursern.com/the-ultimate-list-of-nursing-medical-supplies-and-items-a-new-nurse-student-nurse-needs-to-buy/ Visit our website RegisteredNurseRN.com for free quizzes, nursing care plans, salary information, job search, and much more: http://www.registerednursern.com Check out other Videos: https://www.youtube.com/user/RegisteredNurseRN/videos All of our videos in a playlist: https://www.youtube.com/watch?v=pAhHxt663pU&list=PLQrdx7rRsKfXMveRcN4df0bad3ugEaQnk Popular Playlists: NCLEX Reviews: https://www.youtube.com/playlist?list=PLQrdx7rRsKfWtwCDmLHyX2UeHofCIcgo0 Fluid & Electrolytes: https://www.youtube.com/playlist?list=PLQrdx7rRsKfWJSZ9pL8L3Q1dzdlxUzeKv Nursing Skills: https://www.youtube.com/playlist?list=PLQrdx7rRsKfUhd_qQYEbp0Eab3uUKhgKb Nursing School Study Tips: https://www.youtube.com/playlist?list=PLQrdx7rRsKfWBO40qeDmmaMwMHJEWc9Ms Nursing School Tips & Questions" https://www.youtube.com/playlist?list=PLQrdx7rRsKfVQok-t1X5ZMGgQr3IMBY9M Teaching Tutorials: https://www.youtube.com/playlist?list=PLQrdx7rRsKfUkW_DpJekN_Y0lFkVNFyVF Types of Nursing Specialties: https://www.youtube.com/playlist?list=PLQrdx7rRsKfW8dRD72gUFa5W7XdfoxArp Healthcare Salary Information: https://www.youtube.com/playlist?list=PLQrdx7rRsKfVN0vmEP59Tx2bIaB_3Qhdh New Nurse Tips: https://www.youtube.com/playlist?list=PLQrdx7rRsKfVTqH6LIoAD2zROuzX9GXZy Nursing Career Help: https://www.youtube.com/playlist?list=PLQrdx7rRsKfVXjptWyvj2sx1k1587B_pj EKG Teaching Tutorials: https://www.youtube.com/playlist?list=PLQrdx7rRsKfU-A9UTclI0tOYrNJ1N5SNt Personality Types: https://www.youtube.com/playlist?list=PLQrdx7rRsKfU0qHnOjj2jf4Hw8aJaxbtm Dosage & Calculations for Nurses: https://www.youtube.com/playlist?list=PLQrdx7rRsKfUYdl0TZQ0Tc2-hLlXlHNXq Diabetes Health Managment: https://www.youtube.com/playlist?list=PLQrdx7rRsKfXtEx17D7zC1efmWIX-iIs9
Просмотров: 70665 RegisteredNurseRN
Anne Louise Oaklander | Small Fibers, Big Pain || Radcliffe Institute
 
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As part of the 2017-2018 Epidemics Science Lecture Series at the Radcliffe Institute for Advanced Study, Anne Louise Oaklander presents her lecture "Small Fibers, Big Pain: New Research on Fibromyalgia and Small Fiber Neuropathy," in which she discusses her new findings about unexplained chronic pain-such as that associated with fibromyalgia and Gulf War syndrome-and explains how damage to small nerve endings can lead to chronic fatigue, nausea, and even brain fog. Introduction by Janet Rich-Edwards, faculty codirector of the science program, Radcliffe Institute for Advanced Study; associate professor of medicine, Harvard Medical School; associate professor, Department of Epidemiology, Harvard T.H. Chan School of Public Health For information about the Radcliffe Institute and its many public programs, visit https://www.radcliffe.harvard.edu/.
Просмотров: 9551 Harvard University
Assessment and Management of Patients with Allergic Disorders
 
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Description Not Provided.
Просмотров: 342 Connie Varn
Pyelonephritis - Causes, Clinical Findings, Treatment
 
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http://usmlevideolectures.blogspot.com/
Просмотров: 61379 USMLEVideoLectures
Immunology: Epitopes
 
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1976 copyright act entitles fair use for educational and non profit purposes, my videos aren’t monetized, and the DMCA extends the definition from the 1976 copyright cat
Просмотров: 11812 Thomas Underhill
Colorectal (Colon) Cancer Screening Guidelines *USMLE STEPs 1, 2 & 3*
 
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Super high-yield Colorectal Cancer Screening Guidelines and Colonoscopy after Polypectomy Screenings for the USMLE STEP exams as well as for your clinical practice. Good luck with your studies! About the USMLE CHANNEL - We are Niya and Raj, and we are here to share with you our passion for Medicine! We have successfully passed all USMLE STEPs and we know what YOU NEED to know, in order to SCORE phenomenally HIGH and enjoy RESIDENCY in the UNITED STATES. We are creating medical content videos for all STEPs - yes ALL of them-1, 2 and 3! Our videos are finely tuned, to provide you with the complete understanding of the most updated material, that will help you pick the right answers on the tests and in your future clinical practice. Because what you are studying here, trust us, you will be needing during your residency training! In our videos, we will be using our very own PERSONALLY CRAFTED MIND MAPS, that will make all that large content seem small and make concepts stick. We'll also be integrating quite a lot, which in our years of teaching experience has proven to give the best results. We have walked your journey, and if when starting it, we knew what we know now, life would have been so much easier. And that’s the idea behind the birth of this channel, to be here for you and give you the knowledge that will help you succeed. Enjoy our videos! Stay updated! Love what you are learning! Because in the end - IT IS ALL WORTH IT! DISCLAIMER: The Video Content has been made available for informational and educational purposes only. The Video Content is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Always seek the advice of qualified health professionals and educators for clarification or continued education on any materials expressed herein prior to patient treatment and skills implementation. Attributions: Music -https://www.bensound.com Photo by Aaditya Arora from Pexels
Просмотров: 618 THE USMLE CHANNEL.
The impact of primary Sjögren’s syndrome on female sexual function
 
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Jaap van Laar talks to Jolien F. van Nimwegen about her article: ‘The impact of primary Sjögren’s syndrome on female sexual function’ http://rheumatology.oxfordjournals.org/content/54/7/1286.full © Oxford University Press
Просмотров: 871 Oxford Academic (Oxford University Press)
Sjogren's Syndrome Conference: Ann Fry Keynote Speaker- Hyatt Regency, Bethesda MD (Long Version)
 
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Sjogren's Syndrome Annual Patient Conference: Ann Fry Keynote Speaker- Hyatt Regency, Bethesda MD (Long Version). Ann was the keynote speaker to help patients embrace illness and still have a thriving life.
Просмотров: 1224 Ann Fry
10. Mononuclear Phagocytes (Kelley and Firestein’s Textbook of Rheumatology 10th Edition-2017)
 
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Kelley and Firestein’s Textbook of Rheumatology 10th Edition-2017 1 Biology of the Normal Joint 2 Synovium 3 Cartilage and Chondrocytes 4 Biology, Physiology, and Morphology of Bone 5 Muscle 6 Biomechanics 7 Regenerative Medicine and Tissue Engineering 8 Proteinases and Matrix Degradation 9 Dendritic Cells 10 Mononuclear Phagocytes 11 Neutrophils 12 T Lymphocytes 13 B Cells 14 Fibroblasts and Fibroblast-like Synoviocytes 15 Mast Cells 16 Platelets 17 Innate Immunity 18 Adaptive Immunity and Organization of Lymphoid Tissues 19 Autoimmunity 20 Metabolic Regulation of Immunity 21 Genetics of Rheumatic Diseases 22 Epigenetics of Rheumatic Diseases 23 Complement System 24 Prostaglandins, Leukotrienes, and Related Compounds 25 Cell Recruitment and Angiogenesis 26 Cytokines 27 Principles of Signaling 28 Immunologic Repercussions of Cell Death 29 Experimental Models for Rheumatoid Arthritis 30 Neuronal Regulation of Pain and Inflammation 31 Clinical Research Methods in Rheumatic Disease 32 Economic Burden of Rheumatic Diseases 33 Assessment of Health Outcomes 34 Biologic Markers in Clinical Trials and Clinical Care 35 Occupational and Recreational Musculoskeletal Disorders 36 Cardiovascular Risk in Inflammatory Rheumatic Disease 37 Cancer Risk in Rheumatic Diseases 38 Introduction to Physical Medicine, Physical Therapy, and Rehabilitation 39 Pregnancy and Rheumatic Diseases 40 History and Physical Examination of the Musculoskeletal System 41 Acute Monoarthritis 42 Evaluation and Differential Diagnosis of Polyarthritis 43 Skin and Rheumatic Diseases 44 The Eye and Rheumatic Diseases 45 Neck Pain 46 Shoulder Pain 47 Low Back Pain 48 Hip and Knee Pain 49 Foot and Ankle Pain 50 Hand and Wrist Pain 51 Temporomandibular Joint Pain 52 Fibromyalgia 53 Synovial Fluid Analyses, Synovial Biopsy, and Synovial Pathology 54 Arthrocentesis and Injection of Joints and Soft Tissue 55 Anti-nuclear Antibodies 56 Autoantibodies in Rheumatoid Arthritis 57 Acute Phase Reactants and the Concept of Inflammation 58 Imaging in Rheumatic Diseases 59 Biology and Therapeutic Targeting of Prostanoids 60 Glucocorticoid Therapy 61 Traditional DMARDs 62 Immunosuppressive Drugs 63 Anti-cytokine Therapies 64 Cell-Targeted Biologics and Emerging Targets 65 Novel Intra-cellular Targeting Agents in Rheumatic Disease 66 Urate-Lowering Therapy 67 Analgesic Agents in Rheumatic Disease 68 Nutrition and Rheumatic Diseases 69 Etiology and Pathogenesis of Rheumatoid Arthritis 70 Clinical Features of Rheumatoid Arthritis 71 Treatment of Rheumatoid Arthritis 72 Early Synovitis and Early Undifferentiated Arthritis 73 Sjögren’s Syndrome 74 Etiology and Pathogenesis of Spondyloarthritis 75 Ankylosing Spondylitis 76 Undifferentiated Spondyloarthritis 77 Psoriatic Arthritis 78 Enteropathic Arthritis 79 Etiology and Pathogenesis of Systemic Lupus Erythematosus 80 Clinical Features of Systemic Lupus Erythematosus 81 Treatment of Systemic Lupus Erythematosus 82 Anti-phospholipid Syndrome 83 Etiology and Pathogenesis of Scleroderma 84 Clinical Features and Treatment of Scleroderma 85 Inflammatory Diseases of Muscle and Other Myopathies 86 Overlap Syndromes 87 Classification and Epidemiology of Systemic Vasculitis 88 Giant Cell Arteritis, Polymyalgia Rheumatica, and Takayasu’s Arteritis 89 Anti-neutrophil Cytoplasmic Antibody?Associated Vasculitis 90 Polyarteritis Nodosa and Related Disorders 91 Immune Complex?Mediated Small-Vessel Vasculitis 92 Primary Angiitis of the Central Nervous System 93 Behçet’s Disease 94 Etiology and Pathogenesis of Hyperuricemia and Gout 95 Clinical Features and Treatment of Gout 96 Calcium Crystal Disease 97 Familial Autoinflammatory Syndromes 98 Pathogenesis of Osteoarthritis 99 Clinical Features of Osteoarthritis 100 Treatment of Osteoarthritis 101 Metabolic Bone Disease 102 Proliferative Bone Diseases 103 Osteonecrosis 104 Relapsing Polychondritis 105 Heritable Diseases of Connective Tissue 106 Etiology and Pathogenesis of Juvenile Idiopathic Arthritis 107 Clinical Features and Treatment of Juvenile Idiopathic Arthritis 108 Pediatric Systemic Lupus Erythematosus, Juvenile Dermatomyositis, Scleroderma, and Vasculitis 109 Bacterial Arthritis 110 Lyme Disease 111 Mycobacterial Infections of Bones and Joints 112 Fungal Infections of Bones and Joints 113 Rheumatic Manifestations of Human Immunodeficiency Virus Infection 114 Viral Arthritis 115 Rheumatic Fever and Post-streptococcal Arthritis 116 Amyloidosis 117 Sarcoidosis 118 Hemochromatosis 119 Hemophilic Arthropathy 120 Rheumatic Manifestations of Hemoglobinopathies 121 IgG4-Related Disease 122 Arthritis Accompanying Endocrine and Metabolic Disorders 123 Musculoskeletal Syndromes in Malignancy 124 Tumors and Tumor-like Lesions of Joints and Related Structures
Просмотров: 237 Tâm Nguyễn Trung
Peripheral Vascular Disease
 
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Peripheral Vascular Disease is a lecture capture video for Delaware Tech's Associate Degree Nursing program (NUR 190 course). This product was funded by a grant awarded by the U.S. Department of Labor’s Employment and Training Administration. The product was created by the grantee and does not necessarily reflect the official position of the U.S. Department of Labor. The Department of Labor makes no guarantees, warranties, or assurances of any kind, express or implied, with respect to such information, including any information on linked sites and including, but not limited to, accuracy of the information or its completeness, timeliness, usefulness, adequacy, continued availability, or ownership. Unless otherwise specified, this work by Delaware Technical Community College is licensed under a Creative Commons Attribution 4.0 International License: http://creativecommons.org/licenses/by/4.0/.
Просмотров: 200 Delaware Tech Associate Degree Nursing
Low Back Pain -   Disc  Herniation ,Sciatica  - Everything You Need To Know - Dr. Nabil Ebraheim
 
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Dr. Ebraheim’s educational animated video describing disc herniation of the lumbo sacral spine, the etiology, signs and symptoms, diagnosis , and treatment options. The spine is comprised of bony vertebrae separated by discs. Spinal cord ends at level of T12-L1. Conus Medullaris is the lower end of the spinal cord. Cauda Equina is a division of multiple nerve roots beginning at the level of L1. Conditions of the lumbar spine including disc herniation are a main cause of low back pain. The lumbar spine (lower back) consists of five vertebrae numbered L1 to L5. These vertebrae are attached to the sacrum at the lower end of the spine. The discs between the vertebrae are round cushioning pads which act as shock absorbers. Inner disc layer is soft gelatinous tissue (Nucleus Pulposus). Outer disc layer is thick and strong tissue (Annulus Fibrosus). A disc herniation of the lumbosacral region could involve the nerve roots. In 95% of all disc herniation cases, the L4-L5 or L5-S1 disc levels are involved. Herniation of the L4-L5 disc will affect the L5 nerve root. herniation of the L5-S1 disc will affect the S1 nerve root. Types of disc herniation include: 1-Protruision/Bulge: a bulging disc with intact annular and posterior longitudinal ligament fibers. 2-Disc herniation: Type A •Disruption of inner annular fibers with intact outer annular fibers. Type B •Disrupted annulas with tail of disc material extending into the disc space. 3-Sequestration •Free fragment without tail extending into disc space. •Fragment may be reabsorbed spontaneously. Typical locations of disc herniation Central •Involves multiple nerve roots. •Predominantly cause low back pain more than leg pain •May cause incontinence of the bladder and bowel. •Urgent surgical treatment if patients present with neurological deficits. Posterolateral •Usual location, most commonly involving one nerve root (the lower one). •Example: L4-L5 posterolateral herniation will involve L5 nerve root. Foraminal •Occurs in 8-10% of the cases •Involving the exiting nerve. •Example: L4-L5 foraminal herniation will involve L4 nerve root. Discogenic back pain internal disc disruption •Early disc degeneration. •Pain worsens with flexion/sitting. •Slightly better with extension. •Forward flexion limited on exam. •No radicular symptoms.
Просмотров: 2479242 nabil ebraheim
AGENTS USED IN DYSLIPIDEMIA
 
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Просмотров: 312 AUF Pharmacy 4B
Gait Analysis in Laboratory Animals: Studying Coordinated Movement and Associated Disorders
 
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Gait analysis in lab animals is increasingly being recognized as a key feature in the research of disorders that affect human motor behavior. For example, gait disturbances are symptomatic of patients with Parkinson’s disease, muscular dystrophy and arthritis, but rarely has gait been studied in the animal surrogates. Other assays, such as grip strength, rotarod and activity fall short, both individually and collectively, in describing the convergence of strength, balance and coordination in enabling a subject to ambulate. In this webinar we therefore aim to present key concepts regarding how gait can be studied and its value as a research method for animal models of pain, CNS, neuromuscular and neurodegenerative disorders. In this webinar sponsored by Mouse Specifics, Inc., Dr. Thomas Hampton provides an overview of essential parameters for studying gait in laboratory rodents. Topics include key aspects of coordinated movement [walking] such as stride, swing, stance and ataxia. He discusses how animals execute "stepping" via the convergence of motor and sensory inputs and will present a myriad of conditions that can affect walking along with how these conditions can be quantified for use as physiological markers of movement disorders. Dr. Charles Meshul, Research Biologist and Professor at Veterans Hospital, Portland and Oregon Health & Sciences University, presents gait data obtained from his lab using a progressive animal model of Parkinson’s disease. His group has shown that weekly treatment with the neurotoxin, MPTP, for up to 4 weeks, results in a gradual loss of dopamine within the nigrostriatal pathway. He discusses how this unique model may or may not correlate to the extent of motor function tests available and will discuss the sensitivity of gait analysis in describing mice with Parkinson’s disease. Dr. Carol Milligan, Professor of Neurobiology & Anatomy at Wake Forest School of Medicine, presents behavioral deficits that correlate with early pathology in the SOD1 mouse model of amyotrophic lateral sclerosis (ALS). As Dr. Milligan describes, one potential explanation for why preclinical studies in animal models have not translated positively to clinical trials may be insufficient understanding of when and where pathogenesis begins. Characterization of these early events and correlation to human disease is essential, however, for insight into disease onset, discovery of presymptomatic diagnostic disease markers, and identification of novel therapeutic targets. Dr. Milligan highlights early pathological changes identified in her lab and, importantly, analysis of mouse movement that has revealed deficits corresponding to early pathology. Key Topics: Essential tools and approaches for monitoring and quantifying gait in laboratory rodents Pros and cons of assorted methodologies and measurement approaches (e.g., voluntary vs. overground; incline vs. decline vs. horizontal; features such as negotiating obstacles and responding to visual cues) How various parameters measured relate to the strength, balance, and coordination of the animals How to approach experimental design to ensure data validity
Просмотров: 1051 InsideScientific
OZEN'S criteria for Polyarteritis Nodosa (PAN)
 
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FREE FREE FREE !!! FIGURE1 medical app: Discover medical cases from every specialty their views and advice DOWNLOAD NOW  http://download.figure1.com/greenglobe Prepare for USMLE,UK,CANADIAN,AUSTRALIAN, NURSING & OTHER MEDICAL BOARD examinations around the globe with us.Understand the basics, concepts and how to answer wisely and score 99 in each step. we are here to help you. What are you waiting for subscribe now!!! SUBSCRIBE NOW: http://bit.ly/161OmbF For Business inquiries: allornonelaw4business@gmail.com Join our USMLE step 1 prep Zone : https://www.facebook.com/groups/730000020375744 Join our USMLE CK STUDY GROUP: https://www.facebook.com/groups/320959178079398
Просмотров: 2343 allornonelaw
Gut reactions: host microbiome interactions in the intestine in health and disease
 
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Gut reactions: host microbiome interactions in the intestine in health and disease Air date: Wednesday, March 14, 2018, 3:00:00 PM Category: WALS - Wednesday Afternoon Lectures Runtime: 01:00:44 Description: NIH Director's Wednesday Afternoon Lecture Series The gastrointestinal tract is home to a large number and vast array of bacteria that play an important role in nutrition, immune-system development, and host defense. In inflammatory bowel disease there is a breakdown in this mutualistic relationship resulting in aberrant inflammatory responses to intestinal bacteria. Studies in model systems indicate that intestinal homeostasis is an active process involving a delicate balance between effector and immune suppressive pathways. For her presentation, Dr. Powrie will discuss bacterial pathways that promote intestinal homeostasis and host defense, and how these may be harnessed therapeutically. For more information go to https://oir.nih.gov/wals/2017-2018/ Author: Fiona Powrie, D. Phil., Professor; Director, Kennedy Institute of Rheumatology, University of Oxford Permanent link: https://videocast.nih.gov/launch.asp?23754
Просмотров: 802 nihvcast
Lab Findings: Poststreptococcal Glomerulonephritis & Multiple Myeloma - Rapid Review
 
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http://usmlefasttrack.com/?p=5372 Lab, Findings:, Poststreptococcal, Glomerulonephritis, &, Multiple, Myeloma, -, Rapid, Review, Findings, symptoms, findings, causes, mnemonics, review, what is, video, study, Rapid Review, Clinical presenation, First Aid, for, USMLE, Step 1, images, wiki, define, wikipedia, 2013, videos, exam, prep, easy, What is usmle, mnemonic, causes,
Просмотров: 1061 USMLEFastTrack
Sjögren's Solutions - Module 12) Conclusion... important final points
 
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http://www.sjogrens.org.au/solutions 'Sjögren's Solutions' is a collection of 12 video modules about living with Sjögren's Syndrome. Sjögren's is an auto-immune disease that affects the moisture producing glands -- meaning no tears, no saliva and no internal lubrication. The Sjögren's Solutions videos are presented by Beverly Norton who is the founder of The Australian Sjögren's Syndrome Association (TASSA). At the time of her diagnosis in 2000, Bev was the only survivor in Australia of Sjögren's Neuropathy -- 3rd stage Sjögren's -- and she runs TASSA on her own with no government assistance. In many cases Bev and TASSA are the only source of information for patients and also for doctors in Australia who regularly call her for advice. This takes a great toll on Bev who only has limited energy but her quest is to support as many other people as possible. That's where the Sjögren's Solutions video series comes into play. The modules are designed to answer as many preliminary, general questions as possible and take the initial load off Bev. Then, once the modules have been watched, sufferers can choose to contact Bev personally over the phone or via Skype and Join for one-time TASSA membership fee (A$60) and discuss each case in detail and create a unique, tailored approach. Sjögren's is a lifetime disease and by joining TASSA, Bev can continue to offer further personal advice as the disease progresses and symptoms change. But the Sjögren's Solutions modules are TASSA's new first line of information and support, so whether you are new to Sjögren's or moving through the stages, please watch all the videos and explore as many of those solutions as possible and then Join TASSA.
24. Prostaglandins, Leukotrienes, and Related Compounds (Kelley and Firestein’s Textbook of ...)
 
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Kelley and Firestein’s Textbook of Rheumatology 10th Edition-2017 1 Biology of the Normal Joint 2 Synovium 3 Cartilage and Chondrocytes 4 Biology, Physiology, and Morphology of Bone 5 Muscle 6 Biomechanics 7 Regenerative Medicine and Tissue Engineering 8 Proteinases and Matrix Degradation 9 Dendritic Cells 10 Mononuclear Phagocytes 11 Neutrophils 12 T Lymphocytes 13 B Cells 14 Fibroblasts and Fibroblast-like Synoviocytes 15 Mast Cells 16 Platelets 17 Innate Immunity 18 Adaptive Immunity and Organization of Lymphoid Tissues 19 Autoimmunity 20 Metabolic Regulation of Immunity 21 Genetics of Rheumatic Diseases 22 Epigenetics of Rheumatic Diseases 23 Complement System 24 Prostaglandins, Leukotrienes, and Related Compounds 25 Cell Recruitment and Angiogenesis 26 Cytokines 27 Principles of Signaling 28 Immunologic Repercussions of Cell Death 29 Experimental Models for Rheumatoid Arthritis 30 Neuronal Regulation of Pain and Inflammation 31 Clinical Research Methods in Rheumatic Disease 32 Economic Burden of Rheumatic Diseases 33 Assessment of Health Outcomes 34 Biologic Markers in Clinical Trials and Clinical Care 35 Occupational and Recreational Musculoskeletal Disorders 36 Cardiovascular Risk in Inflammatory Rheumatic Disease 37 Cancer Risk in Rheumatic Diseases 38 Introduction to Physical Medicine, Physical Therapy, and Rehabilitation 39 Pregnancy and Rheumatic Diseases 40 History and Physical Examination of the Musculoskeletal System 41 Acute Monoarthritis 42 Evaluation and Differential Diagnosis of Polyarthritis 43 Skin and Rheumatic Diseases 44 The Eye and Rheumatic Diseases 45 Neck Pain 46 Shoulder Pain 47 Low Back Pain 48 Hip and Knee Pain 49 Foot and Ankle Pain 50 Hand and Wrist Pain 51 Temporomandibular Joint Pain 52 Fibromyalgia 53 Synovial Fluid Analyses, Synovial Biopsy, and Synovial Pathology 54 Arthrocentesis and Injection of Joints and Soft Tissue 55 Anti-nuclear Antibodies 56 Autoantibodies in Rheumatoid Arthritis 57 Acute Phase Reactants and the Concept of Inflammation 58 Imaging in Rheumatic Diseases 59 Biology and Therapeutic Targeting of Prostanoids 60 Glucocorticoid Therapy 61 Traditional DMARDs 62 Immunosuppressive Drugs 63 Anti-cytokine Therapies 64 Cell-Targeted Biologics and Emerging Targets 65 Novel Intra-cellular Targeting Agents in Rheumatic Disease 66 Urate-Lowering Therapy 67 Analgesic Agents in Rheumatic Disease 68 Nutrition and Rheumatic Diseases 69 Etiology and Pathogenesis of Rheumatoid Arthritis 70 Clinical Features of Rheumatoid Arthritis 71 Treatment of Rheumatoid Arthritis 72 Early Synovitis and Early Undifferentiated Arthritis 73 Sjögren’s Syndrome 74 Etiology and Pathogenesis of Spondyloarthritis 75 Ankylosing Spondylitis 76 Undifferentiated Spondyloarthritis 77 Psoriatic Arthritis 78 Enteropathic Arthritis 79 Etiology and Pathogenesis of Systemic Lupus Erythematosus 80 Clinical Features of Systemic Lupus Erythematosus 81 Treatment of Systemic Lupus Erythematosus 82 Anti-phospholipid Syndrome 83 Etiology and Pathogenesis of Scleroderma 84 Clinical Features and Treatment of Scleroderma 85 Inflammatory Diseases of Muscle and Other Myopathies 86 Overlap Syndromes 87 Classification and Epidemiology of Systemic Vasculitis 88 Giant Cell Arteritis, Polymyalgia Rheumatica, and Takayasu’s Arteritis 89 Anti-neutrophil Cytoplasmic Antibody?Associated Vasculitis 90 Polyarteritis Nodosa and Related Disorders 91 Immune Complex?Mediated Small-Vessel Vasculitis 92 Primary Angiitis of the Central Nervous System 93 Behçet’s Disease 94 Etiology and Pathogenesis of Hyperuricemia and Gout 95 Clinical Features and Treatment of Gout 96 Calcium Crystal Disease 97 Familial Autoinflammatory Syndromes 98 Pathogenesis of Osteoarthritis 99 Clinical Features of Osteoarthritis 100 Treatment of Osteoarthritis 101 Metabolic Bone Disease 102 Proliferative Bone Diseases 103 Osteonecrosis 104 Relapsing Polychondritis 105 Heritable Diseases of Connective Tissue 106 Etiology and Pathogenesis of Juvenile Idiopathic Arthritis 107 Clinical Features and Treatment of Juvenile Idiopathic Arthritis 108 Pediatric Systemic Lupus Erythematosus, Juvenile Dermatomyositis, Scleroderma, and Vasculitis 109 Bacterial Arthritis 110 Lyme Disease 111 Mycobacterial Infections of Bones and Joints 112 Fungal Infections of Bones and Joints 113 Rheumatic Manifestations of Human Immunodeficiency Virus Infection 114 Viral Arthritis 115 Rheumatic Fever and Post-streptococcal Arthritis 116 Amyloidosis 117 Sarcoidosis 118 Hemochromatosis 119 Hemophilic Arthropathy 120 Rheumatic Manifestations of Hemoglobinopathies 121 IgG4-Related Disease 122 Arthritis Accompanying Endocrine and Metabolic Disorders 123 Musculoskeletal Syndromes in Malignancy 124 Tumors and Tumor-like Lesions of Joints and Related Structures
Просмотров: 39 Tâm Nguyễn Trung
Immunology, 8th Edition
 
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"Immunology, 8th Edition" makes it easy for you to learn all the basic and clinical concepts you need to know for your courses and USMLEs. This medical textbook's highly visual, carefully structured approach makes immunology simple to understand and remember. To learn more, please visit http://www.us.elsevierhealth.com/product.jsp?isbn=9780323080583&dmnum=null&elsca1=CriticalCare&elsca2=soc_med&elsca3=null&elsca4=youtube_ELSpromovideos
Просмотров: 411 Elsevier Medical Books
Glomerular Diseases Part 3/3 (Nephritic Syndrome)
 
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Physiology, Anatomy and Pathology of Glomerular diseases Part 3. High Yield Renal pathology for USMLE Step 1. Renal Glomerular diseases broken down easily. Follow me on twitter for the latest video updates, @daFutureDP_MD. MS2 testing the waters of online teaching. Any suggestions or comments appreciated. Knowledge cannot be bought, it can only be earned. Acquiring it is a process. Applying it is wisdom. Help spread the knowledge, and join the movement. More videos to come.
Просмотров: 2746 David Paul
91. Immune Complex? Mediated Small-Vessel Vasculitis (Kelley and Firestein’s Textbook of ...)
 
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Kelley and Firestein’s Textbook of Rheumatology 10th Edition-2017 1 Biology of the Normal Joint 2 Synovium 3 Cartilage and Chondrocytes 4 Biology, Physiology, and Morphology of Bone 5 Muscle 6 Biomechanics 7 Regenerative Medicine and Tissue Engineering 8 Proteinases and Matrix Degradation 9 Dendritic Cells 10 Mononuclear Phagocytes 11 Neutrophils 12 T Lymphocytes 13 B Cells 14 Fibroblasts and Fibroblast-like Synoviocytes 15 Mast Cells 16 Platelets 17 Innate Immunity 18 Adaptive Immunity and Organization of Lymphoid Tissues 19 Autoimmunity 20 Metabolic Regulation of Immunity 21 Genetics of Rheumatic Diseases 22 Epigenetics of Rheumatic Diseases 23 Complement System 24 Prostaglandins, Leukotrienes, and Related Compounds 25 Cell Recruitment and Angiogenesis 26 Cytokines 27 Principles of Signaling 28 Immunologic Repercussions of Cell Death 29 Experimental Models for Rheumatoid Arthritis 30 Neuronal Regulation of Pain and Inflammation 31 Clinical Research Methods in Rheumatic Disease 32 Economic Burden of Rheumatic Diseases 33 Assessment of Health Outcomes 34 Biologic Markers in Clinical Trials and Clinical Care 35 Occupational and Recreational Musculoskeletal Disorders 36 Cardiovascular Risk in Inflammatory Rheumatic Disease 37 Cancer Risk in Rheumatic Diseases 38 Introduction to Physical Medicine, Physical Therapy, and Rehabilitation 39 Pregnancy and Rheumatic Diseases 40 History and Physical Examination of the Musculoskeletal System 41 Acute Monoarthritis 42 Evaluation and Differential Diagnosis of Polyarthritis 43 Skin and Rheumatic Diseases 44 The Eye and Rheumatic Diseases 45 Neck Pain 46 Shoulder Pain 47 Low Back Pain 48 Hip and Knee Pain 49 Foot and Ankle Pain 50 Hand and Wrist Pain 51 Temporomandibular Joint Pain 52 Fibromyalgia 53 Synovial Fluid Analyses, Synovial Biopsy, and Synovial Pathology 54 Arthrocentesis and Injection of Joints and Soft Tissue 55 Anti-nuclear Antibodies 56 Autoantibodies in Rheumatoid Arthritis 57 Acute Phase Reactants and the Concept of Inflammation 58 Imaging in Rheumatic Diseases 59 Biology and Therapeutic Targeting of Prostanoids 60 Glucocorticoid Therapy 61 Traditional DMARDs 62 Immunosuppressive Drugs 63 Anti-cytokine Therapies 64 Cell-Targeted Biologics and Emerging Targets 65 Novel Intra-cellular Targeting Agents in Rheumatic Disease 66 Urate-Lowering Therapy 67 Analgesic Agents in Rheumatic Disease 68 Nutrition and Rheumatic Diseases 69 Etiology and Pathogenesis of Rheumatoid Arthritis 70 Clinical Features of Rheumatoid Arthritis 71 Treatment of Rheumatoid Arthritis 72 Early Synovitis and Early Undifferentiated Arthritis 73 Sjögren’s Syndrome 74 Etiology and Pathogenesis of Spondyloarthritis 75 Ankylosing Spondylitis 76 Undifferentiated Spondyloarthritis 77 Psoriatic Arthritis 78 Enteropathic Arthritis 79 Etiology and Pathogenesis of Systemic Lupus Erythematosus 80 Clinical Features of Systemic Lupus Erythematosus 81 Treatment of Systemic Lupus Erythematosus 82 Anti-phospholipid Syndrome 83 Etiology and Pathogenesis of Scleroderma 84 Clinical Features and Treatment of Scleroderma 85 Inflammatory Diseases of Muscle and Other Myopathies 86 Overlap Syndromes 87 Classification and Epidemiology of Systemic Vasculitis 88 Giant Cell Arteritis, Polymyalgia Rheumatica, and Takayasu’s Arteritis 89 Anti-neutrophil Cytoplasmic Antibody?Associated Vasculitis 90 Polyarteritis Nodosa and Related Disorders 91 Immune Complex?Mediated Small-Vessel Vasculitis 92 Primary Angiitis of the Central Nervous System 93 Behçet’s Disease 94 Etiology and Pathogenesis of Hyperuricemia and Gout 95 Clinical Features and Treatment of Gout 96 Calcium Crystal Disease 97 Familial Autoinflammatory Syndromes 98 Pathogenesis of Osteoarthritis 99 Clinical Features of Osteoarthritis 100 Treatment of Osteoarthritis 101 Metabolic Bone Disease 102 Proliferative Bone Diseases 103 Osteonecrosis 104 Relapsing Polychondritis 105 Heritable Diseases of Connective Tissue 106 Etiology and Pathogenesis of Juvenile Idiopathic Arthritis 107 Clinical Features and Treatment of Juvenile Idiopathic Arthritis 108 Pediatric Systemic Lupus Erythematosus, Juvenile Dermatomyositis, Scleroderma, and Vasculitis 109 Bacterial Arthritis 110 Lyme Disease 111 Mycobacterial Infections of Bones and Joints 112 Fungal Infections of Bones and Joints 113 Rheumatic Manifestations of Human Immunodeficiency Virus Infection 114 Viral Arthritis 115 Rheumatic Fever and Post-streptococcal Arthritis 116 Amyloidosis 117 Sarcoidosis 118 Hemochromatosis 119 Hemophilic Arthropathy 120 Rheumatic Manifestations of Hemoglobinopathies 121 IgG4-Related Disease 122 Arthritis Accompanying Endocrine and Metabolic Disorders 123 Musculoskeletal Syndromes in Malignancy 124 Tumors and Tumor-like Lesions of Joints and Related Structures
Просмотров: 107 Tâm Nguyễn Trung
Lab Findings: Giant Cell Tumor of bone & Membranous Glomerulonephritis
 
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http://usmlefasttrack.com/?p=5381 Lab, Findings:, Giant, Cell, Tumor, of, bone, &, Membranous, Glomerulonephritis, , Findings, symptoms, findings, causes, mnemonics, review, what is, video, study, Rapid Review, Clinical presenation, First Aid, for, USMLE, Step 1, images, wiki, define, wikipedia, 2013, videos, exam, prep, easy, What is usmle, mnemonic, causes,
Просмотров: 607 USMLEFastTrack
Facet Syndrome: What are the Causes and Symptoms?
 
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http://goldcoastchiropractor.com/facet-syndrome-what-are-the-causes-and-symptoms/ Facet Syndrome: What are the Causes and Symptoms? Free yourself from the disturbance brought about by Facet Syndrome. Your Gold Coast chiropractor at Hinterland Chiropractic can provide you with a safe and natural way to effectively eliminate the pain and discomfort using a holistic approach. Hinterland Chiropractic Address: 7 Price Street, Nerang, QLD 4211 Phone: (07) 5527 3133 Webstie: http://goldcoastchiropractor.com Google Maps: https://www.google.com/maps/place/Hinterland+Chiropractic/@-27.988451,153.336957,15z/data=!4m2!3m1!1s0x0:0x927483ec31ce218b?hl=en-US
Просмотров: 153 Hinterland Chiropractic
Type III Hypersensitivity  (immune complex mediated)
 
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Please encourage us to like to our videos
Просмотров: 49 Department of Biology
Transparency, consumerism, and value in orthopedics: Definitions and implications
 
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Transparency, consumerism, and value in orthopedics defines the central concepts of healthcare transparency and their implications for practicing orthopedists. Visit http://www.ccfcme.org/highvaluevideo to claim CME credit or learn more about the Transparency, consumerism, and value in orthopedics series. The video was produced by the Cleveland Clinic Foundation Center for Continuing Education and Castlight Health. Interested in related CME education? Visit http://www.clevelandclinicmeded.com/specialties/Orthopaedics.aspx?id=137&name=Orthopaedics
Просмотров: 87 ClevelandClinicCME